It seems that people who talk about fructose being bad don´t bother to check more info. To view the following links you have to copy and paste the whole text, line by line, to your URL browser.
Take this study for example.
¨We conclude that short-term replacement of other carbohydrate
sources in the diabetic diet with fructose will improve glycemic
control, whereas replacement with sucrose will not aggravate glycemic
control.¨
http://www.ncbi.nlm.nih.gov/pubmed/3783868
Or this one.
¨No significant differences were observed between either the fructose
or the sucrose diet and the control polysaccharide diet in any of the
measures of glycemic control, serum lipid levels, or insulin and C-
peptide secretion.¨
http://www.ncbi.nlm.nih.gov/pubmed/8908389?
ordinalpos=1&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pu
bmed_DiscoveryPanel.Pubmed_Discovery_RA&linkpos=2&log$=relatedarticles
&logdbfrom=pubmed
Or this review.
¨It has been postulated that increasing consumption of fructose may
be a contributory factor in the development of obesity and the
accompanying metabolic abnormalities. Most studies supporting these
hypotheses, however, are animal studies, which suggest that
consumption of high amounts of fructose may stimulate lipogenesis and
thus alter lipid metabolism and increase body weight. This review
explores the effects of dietary fructose on lipid metabolism in
humans, with the conclusion that the data so far do not support any
significant specific adverse effect of fructose apart from its energy
content. A small amount of fructose may even improve glucose
tolerance, and studies to date on diabetic subjects indicate that
isocaloric replacement of some glucose-based carbohydrates with
fructose may improve metabolic control¨
http://journals.sfu.ca/coaction/index.p ... le/1559/14
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This is also interesting. Too much for the idea that fructose by
itself cause obesity. It´s the package of wheat, high-PUFA oils, and
other rubbish what causes overeating, not the fructose by itself.
¨When subjects drank the fructose preload, they subsequently ate
fewer overall calories and fewer grams of fat than when they drank
any of the other preloads¨
http://www.ncbi.nlm.nih.gov/pubmed/2178391
This one shows that starch may neglect fructose´s hability to
satiate hunger.
¨When the preload contained fructose alone as the sole source of
carbohydrate, subjects ate significantly fewer calories and less fat
than when the preload contained glucose alone. When starch was added
to the fructose preload, there was no significant reduction in
calorie and fat intake.¨
http://www.ncbi.nlm.nih.gov/pubmed/1799 ... tsPanel.Pu
bmed_DiscoveryPanel.Pubmed_Discovery_RA&linkpos=1&log$=relatedarticles
&logdbfrom=pubmed